Erin Beattie
Biography
Erin Beattie is the Network Manager for the Neuromuscular Disease Network for Canada (NMD4C), where she leads the coordination of all network activities, fostering collaboration across clinical, research, and advocacy communities. With a deep commitment to strengthening neuromuscular and rare disease capacity in Canada, Erin plays a pivotal role in connecting stakeholders, supporting strategic initiatives, and advancing national programming. She holds a PhD in Laboratory Medicine & Pathobiology from the University of Toronto and an MSc in Pharmacology from Dalhousie University, with research focused on cardiac remodeling in heart failure. Prior to joining NMD4C, she worked as a Research & Development Scientist and Medical Writer at a clinical research organization specializing in respiratory trials, served as a Clinical Science Consultant, and coordinated research at the IBD Centre at the Children’s Hospital of Eastern Ontario (CHEO). Erin is dedicated to building sustainable networks and strengthening Canada’s neuromuscular and rare disease research landscape.
Recent Publications
Osman, H, Masnata, M, Adamji, Z, Rodrigue, X, Nguyen, CÉ, Slayter, J et al.. Strengthening clinical capacity in spinal muscular atrophy: Developing and implementing training on clinical outcome assessments. J Neuromuscul Dis. 2026. 22143602261416298 PMID:41564276
Mortenson, P, Osman, H, Beattie, E, Kagan, C, Larocca, V, Maltais, C et al.. Building capacity for patient-engagement in neuromuscular disease research: A network project. J Neuromuscul Dis. 2025. 22143602251405819 PMID:41400831
Nair, K, Umapathy, K, Farid, T, Masse, S, Mueller, E, Sivanandan, RV et al.. Intramural activation during early human ventricular fibrillation. Circ Arrhythm Electrophysiol. 2011.4 (5)692-703 PMID:21750274
Mueller, EE, Momen, A, Massé, S, Zhou, YQ, Liu, J, Backx, PH et al.. Electrical remodelling precedes heart failure in an endothelin-1-induced model of cardiomyopathy. Cardiovasc Res. 2011.89 (3)623-33 PMID:21062919
Mueller, EE, Grandy, SA, Howlett, SE. Protein kinase A-mediated phosphorylation contributes to enhanced contraction observed in mice that overexpress beta-adrenergic receptor kinase-1. J Pharmacol Exp Ther. 2006.319 (3)1307-16 PMID:16951260
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